Hearing loss is categorized by severity, type, and configuration. Furthermore, a hearing loss may exist in only one ear (unilateral) or in both ears (bilateral). Hearing loss can be temporary or permanent, sudden or progressive. The severity of a hearing loss is ranked according to ranges of nominal thresholds in which a sound must be so it can be detected by an individual. It is measured in decibels of hearing loss, or dB HL. There are three main types of hearing loss: conductive hearing loss, sensorineural hearing loss, and mixed hearing loss.[15] An additional problem which is increasingly recognised is auditory processing disorder which is not a hearing loss as such but a difficulty perceiving sound. The shape of an audiogram shows the relative configuration of the hearing loss, such as a Carhart notch for otosclerosis, 'noise' notch for noise-induced damage, high frequency rolloff for presbycusis, or a flat audiogram for conductive hearing loss. In conjunction with speech audiometry, it may indicate central auditory processing disorder, or the presence of a schwannoma or other tumor.
▶ If inner ear disorders is due to an infection, then medications are prescribed to control or manage the symptoms of the infection. In case the infection is due to a bacterial infection, antibiotics are prescribed to clear it up. For people who are suffering from vertigo due to inner ear disorders, vestibular therapy is often recommended, and is highly effective.
I have had cricket sounds (pulsing noise) in my tinnitus condition for the last 2 months. Supplements help, if the condition includes dizziness. I recommend LipoFlavinoids (or Citrus Flavinoids from other brands like Now), Gingko Bilboa, Tumeric Circumin. As I also have have mild tension headaches I use also B12 - 1000 mcg, B100 complex, Cherry (Bing) extract, B2 - 100mg. If you do not have headaches, I recommend the B100 complex.
Tinnitus is extremely common. Nearly everyone has experienced tinnitus (for example after noisy events or in very quiet environments) 10 per cent of the population experience tinnitus on a regular basis. In about 1-2 per cent of people tinnitus is sufficient to cause distress. However we know that putting anyone in a sufficiently quiet environment will allow them to perceive tinnitus.
Hyperacusis is an increased sensitivity to sound. If you find that everyday or ordinary sounds are uncomfortable, you may have hyperacusis. Whilst it might seem natural to want to block out as much sound as possible, avoiding sound can actually make hyperacusis worse. Talk to your GP about this and ask for a referral to either an ENT Surgeon or Audiovestibular Physician who will be able to suggest management options – often, using sound (in a very controlled way) can improve hyperacusis.
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^ Tyler RS, Pienkowski M, Roncancio ER, Jun HJ, Brozoski T, Dauman N, Dauman N, Andersson G, Keiner AJ, Cacace AT, Martin N, Moore BC (2014). "A review of hyperacusis and future directions: part I. Definitions and manifestations" (PDF). American Journal of Audiology. 23 (4): 402–19. doi:10.1044/2014_AJA-14-0010. PMID 25104073. Archived (PDF) from the original on May 9, 2018. Retrieved September 23, 2017.

The Institute for Occupational Safety and Health of the German Social Accident Insurance has created a hearing impairment calculator based on the ISO 1999 model for studying threshold shift in relatively homogeneous groups of people, such as workers with the same type of job. The ISO 1999 model estimates how much hearing impairment in a group can be ascribed to age and noise exposure. The result is calculated via an algebraic equation that uses the A-weighted noise exposure level, how many years the people were exposed to this noise, how old the people are, and their sex. The model’s estimations are only useful for people without hearing loss due to non-job related exposure and can be used for prevention activities.[98]
Some medications may reversibly affect hearing. These medications are considered ototoxic. This includes loop diuretics such as furosemide and bumetanide, non-steroidal anti-inflammatory drugs (NSAIDs) both over-the-counter (aspirin, ibuprofen, naproxen) as well as prescription (celecoxib, diclofenac, etc.), paracetamol, quinine, and macrolide antibiotics.[63] Others may cause permanent hearing loss.[64] The most important group is the aminoglycosides (main member gentamicin) and platinum based chemotherapeutics such as cisplatin and carboplatin.[65][66]
Deaf culture refers to a tight-knit cultural group of people whose primary language is signed, and who practice social and cultural norms which are distinct from those of the surrounding hearing community. This community does not automatically include all those who are clinically or legally deaf, nor does it exclude every hearing person. According to Baker and Padden, it includes any person or persons who "identifies him/herself as a member of the Deaf community, and other members accept that person as a part of the community,"[114] an example being children of deaf adults with normal hearing ability. It includes the set of social beliefs, behaviors, art, literary traditions, history, values, and shared institutions of communities that are influenced by deafness and which use sign languages as the main means of communication.[115][116] Members of the Deaf community tend to view deafness as a difference in human experience rather than a disability or disease.[117][118] When used as a cultural label especially within the culture, the word deaf is often written with a capital D and referred to as "big D Deaf" in speech and sign. When used as a label for the audiological condition, it is written with a lower case d.[115][116]
Ramirez et al (14) aimed to explore the anatomical and physiological connections in TMD patients with secondary aural symptoms and the central and peripheral mechanisms involved. The authors carried out an extensive peer-reviewed literature search, using data from (12), 436 patients in 49 papers, to analyse aural symptoms (otalgia, tinnitus, vertigo, subjective hearing loss and aural fullness) exacerbated by dysfunctional mouth and jaw dynamics. They proposed a range of muscular, bone communication and neural scenarios to explain this relationship, placing emphasis on tensor tympani muscle involvement and trigeminal nerve dysfunction.
Fluctuating sensorineural hearing loss may be from unknown cause or associated with Ménière’s disease. Symptoms of Meniere’s disease are hearing loss, tinnitus (ringing in the ears), and vertigo. Ménière’s disease may be treated medically with a low-sodium diet, diuretics, and corticosteroids. If the vertigo is not medically controlled, then various surgical procedures are used to eliminate the vertigo.
Sound waves travel through the ear canal to the middle and inner ear, where hair cells in part of the cochlea help transform sound waves into electrical signals that then travel to the brain's auditory cortex via the auditory nerve. When hair cells are damaged — by loud noise or ototoxic drugs, for example — the circuits in the brain don't receive the signals they're expecting. This stimulates abnormal activity in the neurons, which results in the illusion of sound, or tinnitus.
In cases of infection, antibiotics or antifungal medications are an option. Some conditions are amenable to surgical intervention such as middle ear fluid, cholesteatoma, and otosclerosis. If conductive hearing loss is due to head trauma, surgical repair is an option.[5] If absence or deformation of ear structures cannot be corrected, or if the patient declines surgery, hearing aids which amplify sounds are a possible treatment option.[2] Bone conduction hearing aids are useful as these deliver sound directly, through bone, to the cochlea or organ of hearing bypassing the pathology. These can be on a soft or hard headband or can be inserted surgically, a bone anchored hearing aid, of which there are several types. Conventional air conduction hearing aids can also be used.
The ear is one of the most vital sensory organs of the human body. It comprises three major parts: the outer ear, middle ear and inner ear. The outer ear includes the pinna and ear canal. It is separated from the middle ear by an eardrum. The middle ear is an air-filled space present behind the eardrum. The inner ear consists of a system of canals and fluid-filled tube-like structures called labyrinth. There may be various problems associated with the inner ear, which can lead to problems in hearing and balance.
Tinnitus is usually more noticeable in a quiet environment. It’s a bit like candles on a birthday cake – in the lights, the candles aren’t very bright but if you turn the lights off, the candles seem much brighter. With tinnitus, when there is other sound, it doesn’t seem that loud, but when you turn all the other sound off, the tinnitus seems much more noticeable.
Hearing loss may be caused by a number of factors, including: genetics, ageing, exposure to noise, some infections, birth complications, trauma to the ear, and certain medications or toxins.[2] A common condition that results in hearing loss is chronic ear infections.[2] Certain infections during pregnancy, such as cytomegalovirus, syphilis and rubella, may also cause hearing loss in the child.[2][10] Hearing loss is diagnosed when hearing testing finds that a person is unable to hear 25 decibels in at least one ear.[2] Testing for poor hearing is recommended for all newborns.[8] Hearing loss can be categorized as mild (25 to 40 dB), moderate (41 to 55 dB), moderate-severe (56 to 70 dB), severe (71 to 90 dB), or profound (greater than 90 dB).[2] There are three main types of hearing loss: conductive hearing loss, sensorineural hearing loss, and mixed hearing loss.[3]
Sensorineural causes of hearing loss involve the inner ear or brain and are usually, but not always, permanent. Implants and more invasive procedures may help to restore hearing. Birth defects (prenatal infection, injury during childbirth, and genetic disorders), infections particularly in childhood and age-related degeneration (presbycusis / presbyacusis) are more frequent causes of this type of hearing loss. Sudden, unilateral hearing loss may be related to conditions like a stroke.
3. A number of headset manufacturers produce “acoustic shock prevention” devices for their headsets to plug into. They are relatively primitive devices that simply attenuate (raise or lower volume of) incoming sound. This means that if a loud noise comes in the unit will suppress all sound that the headset wearer hears including the caller’s voice.

Outer ear infection: otitis externa – usually affects adults aged 45 to 75. It affects the ear canal and is often caused by bacterial infection of the skin of the canal, or a fungus or a yeast. It can also be caused by an irritation such as wearing earplugs or a hearing aid. It is common in people who suffer from skin problems such as eczema, psoriasis or dermatitis but also in people who are keen swimmers.


This is a meditation technique that is used frequently for pain management, and more recently for tinnitus. The idea is that we tend to resist unpleasant sensations (eg hearing tinnitus). If we stop resisting and allow the unpleasant sensation, this alters our awareness to include more sensations. We start to notice that sensations become less dominant once our attention moves away from them and focuses on a different part of the body. All of this can change in a moment, simply by changing our awareness. If we use mindfulness effectively, we can create some space from the tinnitus and in that space, we can decide how we’re going to respond to it. It’s a wonderful way of achieving ‘peace and quiet’.
If you are experiencing hearing loss, you should see an ENT (ear, nose, and throat) specialist, or otolaryngologist, who can make a specific diagnosis for you, and talk to you about treatment options, including surgical procedures. A critical part of the evaluation will be a hearing test (audiogram) performed by an audiologist (a professional who tests hearing function) to determine the severity of your loss as well as determine if the hearing loss is conductive, sensorineural, or a mix of both.
Disorders responsible for hearing loss include auditory neuropathy,[57][58] Down syndrome,[59] Charcot–Marie–Tooth disease variant 1E,[60] autoimmune disease, multiple sclerosis, meningitis, cholesteatoma, otosclerosis, perilymph fistula, Ménière's disease, recurring ear infections, strokes, superior semicircular canal dehiscence, Pierre Robin, Treacher-Collins, Retinitis Pigmentosa, Pedreds, and Turners syndrome, syphilis, vestibular schwannoma, and viral infections such as measles, mumps, congenital rubella (also called German measles) syndrome, several varieties of herpes viruses,[61] HIV/AIDS,[62] and West Nile virus.
Ototoxic drugs also may cause subjective tinnitus, as they may cause hearing loss, or increase the damage done by exposure to loud noise. Those damages may occur even at doses that are not considered ototoxic.[28] More than 260 medications have been reported to cause tinnitus as a side effect.[29] In many cases, however, no underlying cause could be identified.[2]
Conductive hearing loss makes all sounds seem faint or muffled. The hearing loss is usually worse in lower frequencies. Congenital conductive hearing loss is identified through newborn hearing screening or may be identified because the baby has microtia or other facial abnormalities. Conductive hearing loss developing during childhood is usually due to otitis media with effusion and may present with speech and language delay or difficulty hearing. Later onset of conductive hearing loss may have an obvious cause such as an ear infection, trauma or upper respiratory tract infection or may have an insidious onset related to chronic middle ear disease, otosclerosis or a tumour of the naso-pharynx. Earwax is a very common cause of a conductive hearing loss which may present suddenly when the wax blocks sound from getting through the external ear canal to the middle and inner ear.
Look into biofeedback therapy for your tinnitus. If you are depressed, stressed, or fatigued, then you may be more susceptible to normal head sounds. Look into biofeedback therapy from a counselor who can help you to tune into the feelings and situations that cause or worsen your tinnitus. This may help you to stop tinnitus when it starts and prevent it from coming back.[2]
HSE considers that, in general, call handlers' daily personal noise exposure is unlikely to exceed the 80 dB lower exposure action value defined in the Control of Noise at Work Regulations 2005, provided good practice in the management of noise risks is followed. Call handlers should be encouraged to report to management exposure to acoustic shock incidents and management should keep a record of these reported events.
Noise is widely recognized as an occupational hazard. In the United States, the National Institute for Occupational Safety and Health (NIOSH) and the Occupational Safety and Health Administration (OSHA) work together to provide standards and enforcement on workplace noise levels.[90][91] The hierarchy of hazard controls demonstrates the different levels of controls to reduce or eliminate exposure to noise and prevent hearing loss, including engineering controls and personal protective equipment (PPE).[92] Other programs and initiative have been created to prevent hearing loss in the workplace. For example, the Safe-in-Sound Award was created to recognize organizations that can demonstrate results of successful noise control and other interventions.[93] Additionally, the Buy Quiet program was created to encourage employers to purchase quieter machinery and tools.[94] By purchasing less noisy power tools like those found on the NIOSH Power Tools Database and limiting exposure to ototoxic chemicals, great strides can be made in preventing hearing loss.[95]
The Institute for Occupational Safety and Health of the German Social Accident Insurance has created a hearing impairment calculator based on the ISO 1999 model for studying threshold shift in relatively homogeneous groups of people, such as workers with the same type of job. The ISO 1999 model estimates how much hearing impairment in a group can be ascribed to age and noise exposure. The result is calculated via an algebraic equation that uses the A-weighted noise exposure level, how many years the people were exposed to this noise, how old the people are, and their sex. The model’s estimations are only useful for people without hearing loss due to non-job related exposure and can be used for prevention activities.[98]
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HSE considers that, in general, call handlers' daily personal noise exposure is unlikely to exceed the 80 dB lower exposure action value defined in the Control of Noise at Work Regulations 2005, provided good practice in the management of noise risks is followed. Call handlers should be encouraged to report to management exposure to acoustic shock incidents and management should keep a record of these reported events.
A 2017 report by the World Health Organization estimated the costs of unaddressed hearing loss and the cost-effectiveness of interventions, for the health-care sector, for the education sector and as broad societal costs.[103] Globally, the annual cost of unaddressed hearing loss was estimated to be in the range of $750–790 billion international dollars.
This is one psychological approach that can be useful in managing tinnitus. The idea is that when you became aware of your tinnitus, you responded to it negatively. For example, you may have thought there was something seriously wrong with your hearing (a belief) and this led to you being anxious (an emotion), and you then tried to feel better, for example by avoiding silence (a behaviour). Some beliefs and behaviours are helpful and that’s great – keep doing them! But some beliefs and/or behaviours are unhelpful and CBT helps you to recognise them, and then you work together with the clinician (usually a psychologist, audiologist or hearing therapist) to find different ways of responding to the tinnitus so it becomes less bothersome.
If you are experiencing hearing loss, you should see an ENT (ear, nose, and throat) specialist, or otolaryngologist, who can make a specific diagnosis for you, and talk to you about treatment options, including surgical procedures. A critical part of the evaluation will be a hearing test (audiogram) performed by an audiologist (a professional who tests hearing function) to determine the severity of your loss as well as determine if the hearing loss is conductive, sensorineural, or a mix of both.
The initial physiological symptoms of acoustic shock are considered to be a direct consequence of excessive, involuntary middle ear muscle contractions. While the stapedial reflex is an acoustic reflex triggered by high volume levels, the tensor tympani reflex is a startle reflex (6, 7) which is exaggerated by high stress levels. The tensor tympani muscle contracts immediately preceding the sounds produced during self-vocalisation, suggesting it has an established protective function to loud sounds (1), assists in the discrimination of low frequency sounds (8), and is involved in velopharyngeal movements (8).
As of 2013 hearing loss affects about 1.1 billion people to some degree.[12] It causes disability in about 466 million people (5% of the global population), and moderate to severe disability in 124 million people.[2][13][14] Of those with moderate to severe disability 108 million live in low and middle income countries.[13] Of those with hearing loss, it began during childhood for 65 million.[15] Those who use sign language and are members of Deaf culture see themselves as having a difference rather than an disability.[16] Most members of Deaf culture oppose attempts to cure deafness[17][18][19] and some within this community view cochlear implants with concern as they have the potential to eliminate their culture.[20] The terms hearing impairment or hearing loss are often viewed negatively as emphasizing what people cannot do, although the terms are still regularly used when referring to deafness in medical contexts.[16][21]
^ Langguth B, Goodey R, Azevedo A, et al. (2007). "Consensus for tinnitus patient assessment and treatment outcome measurement: Tinnitus Research Initiative meeting, Regensburg, July 2006". Tinnitus: Pathophysiology and Treatment. Progress in Brain Research. 166. pp. 525–36. doi:10.1016/S0079-6123(07)66050-6. ISBN 978-0444531674. PMC 4283806. PMID 17956816.
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